Friday, September 13, 2019

Oral Fluoroquinolones and Risk of Mitral and Aortic Regurgitation

Fluoroquinolones (FQs) are one of the most prescribed classes of antibiotics and are favored over other agents for their broad spectrum of antibacterial activity and high oral absorption. In recent years, a number of adverse events have been linked to these drugs. Some of these adverse events include retinal detachment, which has produced mixed results , but others, including aortic aneurysm and dissection , peripheral neuropathy , and cardiac arrhythmias , are more consistent with a causal link with FQs and are now included in a warning from the U.S. Food and Drug Administration (FDA)

Mechanistically, FQs are known to damage connective tissue by inducing oxidative stress within the tendon cells , reducing collagen production , and stimulating the activity of metalloproteinases , all of which may lead to reduction in the integrity of the extracellular matrix . FQs can damage type I and III collagen  that is present in the Achilles tendons , aorta , and aortic valves . The putative chain of pathophysiological events would include FQs that, through their high bioavailability and chelating properties, bind to the collagen leading to eventual degradation of the collagen matrix of the aortic or possibly the mitral valve. This can lead to subsequent aortic or mitral valve regurgitation. This hypothesis has been observed in at least 1 case report where a patient who took ciprofloxacin (750 mg twice daily) for 2 days developed symptoms of decompensation as a result of aortic valve prolapse that, following a cardiac work-up, could only be linked to ciprofloxacin use . The acute onset of collagen damage with FQs has also been shown to lead to rupture of large tendons within hours  and aortic dissection within days .

The results of this study found an association between oral fluoroquinolones and an increased risk of mitral and aortic regurgitation. As such, it might be prudent to consider antibiotics that are chemically distinct to FQs in patients with a previous history of valvular regurgitation who require antibacterial therapy. Future studies are urgently required to confirm or refute these findings

2 comments:

  1. Great post and the use of FQ routinely has certainly decreased in my personal practice due to additional concerns on those patients developing C.Diff associated complications as well. It seems that resistance to the drug class in the community has also risen when looking at antibiograms. We should all heed caution with FQ in light of this as we await further studies on this cardiovascular AE.

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  2. I agree, rapid resistance is a major problem. Certainly in patients with Marfan syndrome I would NOT give this class of antibiotics.

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